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Monitoring updates on XMRV Research for Gulf War Veterans

Gulf War Veterans(90-91) that are dealing with CFS are carefully monitoring what the civilians with CFS are keeping a close eye on and wondering when they will be included in the sample study groups. It is going to be a wait but stay tuned as we monitor any developments on this issue of XMRV findings. Work is occurring quietly but rapidly as far as science goes. Research has very detailed steps to go thru in proving a case but this is getting urgent attention. We at Veterans Today will keep you as updated as possible on this and other issues affecting Gulf War Veterans. So here are two articles from other sources that will help keep you informed.

XMRV Working Group And Nation’s Blood Supply
By Jody Smith
July 28, 2010 – 5:The National Institutes of Health (NIH) formed The Blood XMRV Scientific Research Working Group including the Food and Drug Administration, the Centers for Disease Control and Prevention, blood banks, academic institutions and a patient advocacy organization. The working group is investigating ways to test for the retrovirus XMRV and any possible danger it may present to the blood supply.
Six labs are involved. They are two FDA labs, as well as labs at the National Cancer Institute, the CDC, the Blood Systems Research Institute and the Whittemore-Peterson Institute. These labs have analyzed blood samples containing different amounts of the retrovirus XMRV, in order to determine whether or not the labs’ tests are able to detect XMRV in the blood. This was successful.
“Phase four will analyze blood from 300 blood donors, 25 confirmed XMRV-positive patients, and 30 XMRV-negative samples from 10 independent blood donors. This data will finally get researchers a little closer to answering at least one of the key questions in the still-unfolding drama: Is XMRV widespread in the nation’s blood supply?”

FDA Advisory Committee to Hear About XMRV Working Group’s Research
By Amy Dockser Marcus
Public health officials have been working for months to understand whether the XMRV virus poses a risk to the nation’s blood supply. The concern was sparked by a paper published last year in Science that detected the virus in the blood of 67% of chronic fatigue syndrome patients, compared to just 3.7% of healthy controls.
(Other research has contradicted that finding, and as Health Blog readers know, the scientific controversy is still swirling — at this point, no one knows whether XMRV causes CFS or any other disease — including prostate cancer, to which it has also been linked — or whether the virus or any disease it’s tied to is transmissible through blood infusions. Read this for the latest.)
But based on even the possibility of these connections, and thus a potential threat to the blood supply, the NIH funded a working group that includes the FDA, CDC, blood banks, academic institutions and a patient advocacy organization. (The group includes, it’s worth mentioning, the government scientists whose findings appear to conflict.) The Blood XMRV Scientific Research Working Group is charged with investigating, among other things, the best ways to test for the virus and its prevalence among blood donors.
And at a meeting scheduled for today, members of the FDA’s Blood Products Advisory Committee are slated to hear an informational presentation on XMRV that will include results from the first phase of the working group’s scientific investigation.
In this phase, six labs — two from FDA plus labs at the NCI, the CDC, the Blood Systems Research Institute and the Whittemore-Peterson Institute — used a panel of blood samples spiked with different amounts of XMRV to establish if the labs’ tests are sensitive and reliable enough to detect the virus in blood. They are. All the labs were able to detect at least some amount of XMRV. (Some of the labs have been involved in the dueling research on a link between the virus and CFS.)
The presentation — which the Health Blog previewed ahead of the meeting — suggests that the first phase of the study has some limits. As researchers continue to study XMRV, there is a growing sense that virus taken from patients may be different than the virus the labs studied. That’s why, researchers say, it’s important to start looking at what’s going on in actual people.
That will happen in future phases of the working group’s investigation. In a pilot study of the second phase, WPI collected blood from four CFS patients they had found to be XMRV-positive and sent the samples to CDC and NCI for testing. Results are expected soon. In the third phase, WPI will collect 25 samples from XMRV-positive CFS patients and send them — as well as XMRV-negative samples from healthy people — to all the other labs for testing.
Phase four will analyze blood from 300 blood donors, 25 confirmed XMRV-positive patients, and 30 XMRV-negative samples from 10 independent blood donors. This data will finally get researchers a little closer to answering at least one of the key questions in the still-unfolding drama: Is XMRV widespread in the nation’s blood supply?

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Posted by on Jul 29 2010, With 0 Reads, Filed under Veterans Affairs. You can follow any responses to this entry through the RSS 2.0. Both comments and pings are currently closed.

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4 Comments for “Monitoring updates on XMRV Research for Gulf War Veterans”

  1. I think we need to monitor the happenings of XMRV, but also of CFS which turns Into CFIDS (a viral reactivation subset of CFS) in 60-70% of all CFS Patients. The first thing a patient showing symptoms for (Fatigue, Muscle Weakness, Insomnia, Sleep insomnia and sleep disorders, Joint Pain, Headaches/Migraines, Profound Fatigue, Heart palpitations or chest pain, intolerance to exercise and heat, unfereshed sleep) is to be please tested for HHV-6A, EBC, CMV and the piggy Back co-infections Micoplasma, Chlamydia P (not an STD) and the 5 Lyme Panels. You can read more about these tests at http://vlgonvalcyte.wordpress.com/ Part 2 of this Blog will give specifi test codes as well. These viruses and infections can be treated NOW and then if you test positive for XMRV the retro-virus can be treated when viable testing and treatment methods are available. The co-infections alone (micoplasma) can make you extremely ill, let alone the HHV-6A which is centered in the Brain and Central nervous system.

  2. I think anyone that’s serious about solving this mystery should not only be taking the xmrv side in their investigations but also Associate Professor Donald Staines’s (Queensland Australia) theory of it being a Vasoactive neuropeptide autoimmunity, once you read his book titled “Chronic Fatigue Syndromes and vasoactive nueropeptide autoimmunity” you just might understand how important his hypothesis is and after having accquired this rotten problem 3years, and at the time being extremely active doing all of things we all take for granted. I’ve read this book more times than I care to remember and it is the only plausible theory that really makes sense from every angle and every symptoms and even symptoms I don’t have and this includes Gulf Ware Sydnrome of which just does not sit with the the XMRV theory.

    I will quote a meager amount of this book but I stress it is the most enlightening read I’ve ever come across in my 3years and urge his theory to be taken seriously, because this is one time that I believe being side tracked will unravel something that no one has managed to do so fare. I stress sitting here typing out this is really the last thing I’d want to do if I was feeling the way I did 3 years ago and I understand there are heaps of people out there that have been trying to deal with this for much much longer than me.

    Associate Professor Don Staines quotes that because of their(VN’s, vasoactive nueropeptides) sequence homology, to some extent. with the DNA of viruses and other organisms, it is postulated VNs may be mistaken by the body for invading organisms under certain conditions. This phenomenon is termed ‘molecular mimicry’ although the true extent and validity of the concept generally is still being established.
    VNs are not manufactured and maintained in the same way as the smaller well known neurotransmitters such as acetylcholine (Ach). Unlike these latter transmitters, VNs have no re-uptake mechanism to economise on their usage and once dispersed must be remanufactured and transported to their intended site of action, a protracted and energy consuming process. VNs are also readily broken down by peptidase and possibly antibodies, and therefore they require constant replenishment.
    Compromise of their functions or those of their receptors by any adverse mechanisms imposes and additional impediment to their usual physiological action as considerable time is needed to make up concentration shortfalls. This may account in part for the protracted recovery time seen in people with CFS/ME/FM after exercise or other stressors. As transmitters such as Ach are synergistically engaged with these VNs, co-transmitter functions may be impaired and detrimental effects compounded by VN compromise.

    There is obviously way more in this book and it explains more in depth his theory and how the certain roles of VN’s and how they work synergistically with other things and how they have a huge role in functioning of the heart, immunological, hormonal, protect the brain against excess apoptosis (programmed cell death),neurological roles, VNs modulate higher level CNS executive functions, and much much more,!

    I’d like for the scientific community who are seriously looking at solving this mystery to contact Associate Professor Donald Staines and at the very least chat with him and read his book what harm could this do and who knows what may come of it.

    Cheers

    • Kevin, have you read anything by Byron Hyde, M.D. from Canada regarding Myalgic Encephalomyelitis? He speaks of vascular injury helping to cause M.E. back as far as 1992. He produced an encyclopedia of M.E. and related syndromes back then at the Albany, NY, conference on “CFS”. He had 800 pages of peer reviewed medical articles back then when our own CDC and NIH were still pushing “fatigue lasting more than six consecutive months that kept a person home bound or 50% or more reduced in premorbid activities”.
      our governments activities back then and since then have really been criminal. I am a complete layman in these affairs, but i did suggest as a guest on CNN, On May 4, 1991. that “Larry, many of the sick gulf war vets that are returning home unable to work have symptoms that closely resemble what we have!” After that, Professor Garth Nicolson, who has published more than 400 peer reviewed articles on Cancer and tumor cell biology, produced a 50 question form of some 650 sick Gulf War Veterans, and they had virtually the same answers as our sick M.E. and CFS patients. we were mostly within 5 percentage points of having the same answer. I know that i have pain EVERYWHERE. one doc calls it “autonomic nervous system neuropathy”. but it sure feels like vascular injury to me. don’t know if this makes any sense with Professor Donald Stainers theories, but it might be worth checking out. I gave a speech to the top 636 medical experts in this field back on April 15, 1989, and i said, “until you find SOMETHING, anything,
      that can be replicated by people who do not trust or believe you, then we are all going to be spinning our wheels. Please don’t try to get too esoteric. Just look for a reproducible anomaly,
      and then we will be able to get fair insurance payments, and research money, and respect from your colleagues, and help from our families and friends. We are not sick of being tired. We are TIRED of being Sick. there is a difference. Thanks for bringing up the Professor’s theories. they sound worth pursuing to me.. TMH

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